23 Enero 2015
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Item Type

Journal Article

Title

Ozone contrast media in digital renal angiography. First report in the literature

Author

Ugarte, J C

Author

Wong, R

Author

Alfonzo, J

Author

Banasco, J

Author

Gómez, M

Author

García, J

Author

Menéndez, S

Author

Ladron de Guevara, N

Author

Moreno, E

Author

Román, W

Publication

Nephron

Volume

56

Issue

3

Pages

332

Date

1990

Journal Abbr

Nephron

ISSN

0028-2766

URL

http://www.ncbi.nlm.nih.gov/pubmed/2077419

Accessed

2010-02-24 16:37:47

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NCBI PubMed

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PMID: 2077419

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23 Enero 2015
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Item Type

Journal Article

Title

Ozonated Autohemotherapy in Patients on Maintenance Hemodialysis: Influence on Lipid Profile and

Endothelium

Author

 

Author

Biedunkiewicz, Bogdan

Author

Nieweglowski, Tomasz

Author

Chamenia, Anrzej

Author

Slizien, Alicja

Author

Luty, Joan

Author

Lysiak-Szydlowska, Wieslawa

Author

Rutkowski, Boleslaw

Abstract

Ozonated autohemotherapy (O3-AHT) is used in the treatment of atherosclerotic ischemia of lower

limbs (AILL). The impact of ozone on serum lipids and endothelium injury is of particular interest

since these factors are important in the development of atherosclerotic lesions. To evaluate this issue,

a prospective, placebo-controlled study was designed. Twelve hemodialyzed subjects with AILL

received autohemotherapy with oxygen as a control followed by O3-AHT with ozone concentration of

50 micro g/ml. Serum lipids and plasma activity of von Willebrand factor (vWF) were measured. After

O3-AHT, total cholesterol significantly decreased compared to the baseline (-8.34%) [P < 0.01]. LDL

cholesterol was also significantly lower than the initial value (-17.71%) [P < 0.001]. No significant

changes in the activity of vWF were found after the first session of O3-AHT and after all nine sessions

of O3-AHT. The study demonstrated that O3-AHT did not affect deleteriously the endothelium in

patients with chronic renal failure on maintenance hemodialysis. It may stimulate beneficial changes in

serum lipid profile manifesting as a decrease in the total- and LDL-cholesterol levels.

Publication

Artificial Organs

Volume

28

Issue

2

Pages

234-237

Date

2004

Journal Abbr

Artif Organs

Language

English

ISSN

0160-564X (Print), 1525-1594 (Online)

URL


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PMID: 14961966

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23 Enero 2015
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Item Type

Journal Article

Title

No effects of ozonated autohemotherapy on inflammation response in hemodialyzed patients

Author

Tylicki, Leszek

Author

Biedunkiewicz, Bogdan

Author

Rachon, Dominik

Author

Nieweglowski, Tomasz

Author

Hak, Lukasz

Author

Chamienia, Andrzej

Author

Debska-Slizien, Alicja

Author

Aleksandrowicz, Ewa

Author

Mysliwska, Jolanta

Author

Rutkowski, Boleslaw

Abstract

BACKGROUND: Ozone as a strong oxidant may induce an inflammatory response. AIM: The hypothesis was verified as to whether ozonated autohemotherapy using an ozone dose in therapeutic range changes the plasma concentration of C-reactive protein and interleukin-6, markers of inflammation. METHODS: In a controlled, single-blind, cross-over study, 12 chronically hemodialyzed patients with peripheral arterial disease were exposed to nine sessions of autohemotherapy with blood exposure to oxygen as a control followed by nine sessions of ozonated autohemotherapy with an ozone concentration of 50 microg/ml. RESULTS: There was no statistical difference between C-reactive protein levels at baseline (1.53 +/- 1.01 mg/l), after nine sessions of control autohemotherapy (1.48 +/- 0.96 mg/l), and after nine sessions of ozonated autohemotherapy (1.55 +/- 0.84 mg/l). There was also no statistical difference between the interleukin-6 serum concentration at baseline (438 +/- 118 pg/ml), after nine sessions of control autohemotherapy (444 +/- 120 pg/ml), and after nine sessions of ozonated autohemotherapy (466 +/- 152 pg/ml). CONCLUSION: The results of this study suggest that ozonated autohemotherapy using an ozone concentration of 50 microg/ml does not induce an inflammatory response.

Publication

Mediators of Inflammation

Volume

13

Issue

5-6

Pages

377-380

Date

Dec 2004

Journal Abbr

Mediators Inflamm

DOI

10.1080/09629350400014131

ISSN

0962-9351

URL

http://www.ncbi.nlm.nih.gov/pubmed/15770057

Accessed

2010-02-24 16:42:29

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NCBI PubMed

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PMID: 15770057

 

 

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